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RETURN TO HIGH LEVEL PLAY AFTER ACL RECONSTRUCTION

241

steroid is appropriate. Steroid must not be used

to progress a player towards play however and

is simply rather used to calm the knee down to

allow resolution of effusion for a period of

relatively low loading to regain muscle strength

and neuromuscular control before proceeding

again some weeks later.

In my career cases in which all seemed to be

well apart from an effusion have been

associated with premature graft re-rupture.

Swelling has to be respected.

Factor Three: Aerobic Fitness

It seems obvious that allowing athletes who are

aerobically unfit to play is a risk. They will

fatigue quickly and lose neuromuscular control,

which will put them at risk of graft re-rupture.

Aerobic fitness is easily measured with

functional testing and tests such as a VO

2

Max.

Factor Four: The Concept of Limb

Symmetry

This concept embodies the theory that normal

neuromuscular control in the limb will protect

ACL graft.

Some athletes will have jump/land mechanics

that are putting them at risk of ACL injury.

These need to be corrected during the rehab

process of course. This is particularly true of

female athletes who have a tendency to land

with valgus and external rotation, even in the

uninjured limb.

I explain to the players that they should have

limbs with sufficient muscular symmetry that I

find it hard to judge which was the knee that

was operated upon. They can have their

muscle strength tested with isokinetic testing

and have functional testing such as hop

heights, hop distance and star excursion

measured. Prior to return to play I aim for a

deficit of less than 5% side to side. I also

explain that none of the tests we do are perfect

since they cannot replicate true on-field play.

Nevertheless they, of course, provide helpful

evidence to justify return to play.

Factor Five: Timing

Whilst anecdotally I have had many players

return ahead of schedule I realise this is not

something that should be encouraged since the

majority of players will be at risk if they do this.

Factor Six: Graft Maturation

The reality is that we have no firmunderstanding

of the biology of graft healing. It would be

logical to suggest that the fastest healing within

the bone tunnel would be to blocks of a

quadriceps tendon or patellar tendon. It is

likely that the quality of soft tissue healing in

these grafts is also better as they have a natural

attachment to their bone blocks. It would also

be logical to think that the slowest healing of

all would be withAllograft. The truth is that the

proof of this is lacking. We can only really

guess at the various stages of healing presuming

an initial graft degradation followed by

revascularization and subsequent ligamenti­

sation with the laying down of collagen. The

only judge of the healing process is clinical

examination, the presence or absence of an

effusion, instrumented laxity testing, stress

radiographs, or MRI appearance. MRI scans

report the signal of the graft with a dark black

appearance indicating good healing and a pale

appearance indication oedema in the graft.

Unfortunately we do not have firm data as to

how this can guide is with regards to timing of

return to play. Perhaps a pale graft should delay

return to play and reduce loading durng the

rehabilitation period. When a loose ACL is

obvious by clinical examination, it is too late to

make any adjustments to the graft itself. Adding

a lateral tenodesis in some cases could be

justified, but practically it would be best to

encourage an athlete to accept a slower return

to play hoping that better neuromuscular

control after a longer rehabilitation period

could allow the player to cope dynamically

with their ACL laxity.