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Anterior Knee Pain 3 Months after acl Reconstruction: an International Survey of Practice

387

cases of tendinitis after a failed standard

treatment and within a reasonable delay (after

one year), shockwaves might be helpful but we

are currently lacking evidence.

Isokinetic rehabilitation proved to be of real

interest since it was considered reliable for

67% of our participants. We believe that this

technique improves the standard technique of

cycling by providing better feedback on the

patient’s reactions and it offers the possibility

to use eccentric strengthening programs.

Analgesics and NSAIDs were considered as

standard

for

symptomatic

treatment

(respectively 65 and 63%of reliability) whereas

corticoids, PRP, mesotherapy, conservative

podiatric management, local NSAID were

rarely believed to be effective to relieve anterior

knee pain (oscillating between 19 and 38% of

reliability). The use of cortisone injection and

visco-supplementation is more controversial

but should find a place in cases of cartilage

lesions in the patellofemoral or tibiofemoral

compartment.

We believe that the “tendinitis” should no

longer be used for all disorders related to

anterior knee pain. The physician needs to

differentiate structural or anatomical lesions

from functional problems. To do so, the first

step is to obtain objective findings such as

postoperative plain X-ray pictures to look for

patellofemoral dysplasia (N. Bonin reported

15% of dysplasia in the population of ruptured

ACLcompared to3%in the general population),

fractures, osteopenia (reflex sympathetic

dystrophy), patella bipartita, tunnel mal­

positioning, type 2 Patella in the Grelsamer

classification [1] (the “big nose” that could be

a cause of tendinitis by impingement).

Furthermore, MRI should be performed to

identify the precise origin of the pain that could

be inside the patellofemoral compartment or be

an anterior projection of a femorotibial

compartment disorder. In the patellofemoral

compartment, one might look for cartilage

edema, tendinitis, Hoffa syndrome, Cyclops

syndrome, synovitis, arthrofibrosis, or bone

bruising (condyles or tibial tunnel) [2]. The

femorotibial compartments need to be screened

as well for cartilage or meniscal lesions. If no

structural lesion can be seen on the MRI and if

a neuropathic or neurological problem (such as

neuroma of infrapatellar branches of the

saphenous nerve) is excluded by the DNS4

score, functional disorders might be

investigated with an isokinetic evaluation

(fig. 3). After 3 postoperative months, flexor

insufficiency often jeopardizes the jogging

phase. After 6 postoperative months, it is

possible to assess ratios between extensor and

flexor muscles, which need to be well balanced,

with deficits lower than 15% to avoid pain or a

new injury. Condouret showed that there was a

relationship between the level of extensor and

flexor recovery and the quality of functional

results with minimal muscle deficits close to

5% if the IKDC score was over 90 and deficits

falling to 15% in the group with IKDC score

less than 90 [3].

Once the diagnosis is accurately made, an

adapted treatment is possible in addition to

cryotherapy, NSAIDs, and painkillers. If

cycling and strengthening programs remain the

gold standard for patellofemoral pain with or

without signs of dysplasia, we learned from

E. Arendt, that core strengthening was equally

Fig. 3: Anterior knee pain due to extensor

insufficiency. Isokinetic assessment.